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Inhibiting BACE1 to reverse synaptic dysfunctions in Alzheimer's disease - ScienceDirect
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Novel targets in Alzheimer's disease: A special focus on microglia - ScienceDirect
Memory Impairment in Transgenic Alzheimer Mice Requires Cellular Prion Protein | Journal of Neuroscience
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PDF) Alzheimer's disease as a syndrome of overloaded amyloidic synaptic tagging in memory networks
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PDF) Alzheimer's disease as a syndrome of overloaded amyloidic synaptic tagging in memory networks
Bioinorganic Chemistry of Alzheimer's Disease | Chemical Reviews
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Peptides, Peptidomimetics, and Carbohydrate–Peptide Conjugates as Amyloidogenic Aggregation Inhibitors for Alzheimer's Disease | ACS Chemical Neuroscience
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Memory Impairment in Transgenic Alzheimer Mice Requires Cellular Prion Protein | Journal of Neuroscience
Ablation of Cellular Prion Protein Does Not Ameliorate Abnormal Neural Network Activity or Cognitive Dysfunction in the J20 Line of Human Amyloid Precursor Protein Transgenic Mice | Journal of Neuroscience
Inhibiting BACE1 to reverse synaptic dysfunctions in Alzheimer's disease - ScienceDirect
Inhibiting BACE1 to reverse synaptic dysfunctions in Alzheimer's disease - ScienceDirect
The complexity of Alzheimer's disease: an evolving puzzle | Physiological Reviews
Defining the Neuropathological Aggresome across in Silico, in Vitro, and ex Vivo Experiments | The Journal of Physical Chemistry B
PDF) Exosomes neutralize synaptic-plasticity-disrupting activity of Aβ assemblies in vivo